CXCL10 deficiency limits macrophage infiltration, preserves lung matrix, and enables lung growth in bronchopulmonary dysplasia

Table 1 Clinical data of lungs of infants with and without bronchopulmonary dysplasia (BPD)

Healthy human sample
Donor ID	Calculated age (months)	Sex	Race	ClinPathDx: control
D031	7.64	Male	White	Normal growth and structure
D090	14.70	Female	NR	Normal growth and structure
D046	36.5	Male	White	Normal growth and development
D139	47.67	Female	One race	Normal lung growth and structure
D056	68.21	Male	White	Normal growth and development
BPD human sample
Donor ID	Calculated age (months)	Sex	Race	ClinPathDx: BPD/CLD
D086	8.94	Male	White	25 weeks at birth, probable BPD with respiratory failure
D141	13.40	Male	White	25 weeks at birth, BPD; moderate deficient alveolarization, chronic inflammation, airway muscle hyperplasia, medial hypertrophy of small arteries
D039	39.13	Female	NR	23 weeks at birth, “new” BPD
D053	37.87	Male	White	24 weeks at birth, BPD, mild-moderate bronchiolitis
D055	59.96	Male	White	32 weeks at birth, possible mild BPD, asthma, moderate bronchopneumonia

NR not reported, CLD chronic lung disease, ClinPathDx clinical pathology diagnostic

ISSN: 1880-8190